high yield revision - abinash_singh

[ArchivalUser]

please dont reply...only reply if some mistakes are there.....you may add threads to it....some high yield

[ArchivalUser]

PROSTAGLANDIN INHIBITION IS KEY TO CONTROL OF PRIMARY DYSMENNORRHEA / NSaID SUCH AS IBUPROFEN

--PRIMARY DYSMENORRHEA IS PAIN WITH MENSTRUATION IN ABSENCE OF PELVIC PATHOLOGY

-INHIBITION OF OVULATION WITH ORAL CONTRACEPTIVE , DEPOT MEDROXYPROGESTERONE ACETATE OR A GONDOTROPHIC HORMONE AGONIST IS ALSO EFFECTIVE.

-ORAL CONTRACEPTIVE DECREASE PROSTAGLANDIN SYNTHESIS BY CAUSING ATROPHY AND DECIDUALIZATION OF THE ENDOMETRIUM , RELEF OF DYS MENORRHEA MAY BE ENHANCED BY ORAL CONTRACEPTIVE AND AN NSAID TAKEN ON SCHEDULED BASIS THAT IS NSAID IS TAKEN CONTINUOUSLY ON SCHEDULE DURING THOSE DAYS.

ALTHOUGH FLUOXETINE OR OTHER SELECTIVE SEROTININE UPTAKE INHIBITOR ARE USEFUL FOR PREMENSTRUAL SYNDROME OR THE PREMENSTRUAL DYSMORPHIC DISORDER PMDD THESE ARE INEFFECTIVE IN RELIEVING DYSMENORRHEA

prostaglandin also cause bowel to contract....so associated with diarhhoea