01-15-2014, 01:43 PM
In order to investigate possible tx options for ARDS, experimental animals are anesthetized and subjected to smoke inhalation (cotton smoke) according to well established experimetnal protocol. Experimental animals are given compounds designed to upregulate specific mediators involed in inflammatory cascade. Upregulation of which mediator would most likely prevent or amelilorate lung injury induced in this experimental model of ARDS?
a. IL 1
b. IL 10
c. NFKB
d. Toll like receptors
e. TNF
ans is A but I dont get why...the question is asking what would present lung injury..IL1 would cause more injury right? its increased in inflammation..Acute phase rectant
could someone plz explain this? and why the other choices are wrong?? plz thank u
a. IL 1
b. IL 10
c. NFKB
d. Toll like receptors
e. TNF
ans is A but I dont get why...the question is asking what would present lung injury..IL1 would cause more injury right? its increased in inflammation..Acute phase rectant
could someone plz explain this? and why the other choices are wrong?? plz thank u