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NBME#2 block 2 Q1-----------Q50 - maryam2009
#31
@ amusmle can u explain regarding this microscopy of myxoma--why A.
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#32
@sodium

I'm sorry, my mistake, the question was 46 from NBME2, Block 1
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#33
A,myxoma
a neoplasm composed of stellate to plump cytologically bland mesenchymal cells set in a myxoid stroma.
cytologically bland mesenchymal cells, so-called “myxoma” or “lepidic” cells .
Myxoma cells are frequently stellate with eosinophilic cytoplasm and indistinct cell borders. Their ovoid nuclei are typically pale with open chromatin. Nucleoli may be prominent.



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#34
Q25?
please someone post the answer with the explanation,thank you
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#35
26.A
CMV...infection in immunosuppressed patients,transplant rcipients.
most common viral cause of mental retardation, Congenital defects ..deafness ,petechia,hepatosplenomegaly,macroencephaly,seizure....bluberrymuffin baby
mononucleosis(negative monospot

infected cells have Owl's eye appearance
trethment:Gancyclovir
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#36
27.E
clinical manifestation...hematuria....
associated with paraneoplastic syndrome due to the ectopicEPO, ACTH,PTHrP and prolactin
Gross....golden -yellow tumorin the midkidney with areas hemorrage and necrosis
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#37
@maryam
Q27. Isn't F?
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#38
28.B
The random X-inactivation which occurs early in a developing female can result in skewed or non-random X-inactivation for a patch, tissue or organ.
During the process of X-inactivation one of the female's two X chromosome is inactivated.
This becomes important in medicine when a carrier female of an X-linked recessive condition 'randomly' inactivates the chromosome containing the unaffected gene. This can lead to mild symptoms of the disease
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#39
25?
I don't know the answer,but in enclosed response, it is G
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#40
29.C

Fibroblast Growth Factor (FGF) Macrophages, mast cells, T-lymphocytes Promotes angiogenesis, granulation, and epithelialization via endothelial cell, fibroblast, and keratinocyte migration, respectively.
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