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Ajeet: The concept of all somatosensory modalities is controlled by somatosensory cortex in the postcentral gyrus which is affected.
the elementary somatosensory functions, which consist of light touch, pain, thermal sensation, joint position sense, and vibration sense, and the intermediate somatosensory functions, which include 2-point discrimination, tactile localization, weight, texture, and shape perception.
http://www.springerlink.com/(ummnqz45pqf...rer=parent&backto=issue,10,34;journal,63,985;linkingpublicationresults,1:100440,1
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" graphesthesiae, two point discrimination, tactile localisation " you described are intermidiate functions. you have missed the main primary functions of the cortex which are described in the question.
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IMP points that is controversial to what you have written are: considering this is purely algesic lesion of thalamus, limb ataxia is not seen( hemiplegic gait is not ataxia), minimal weakness can be seen in thalmic lesion, no persistent spontaneous pain in this patient, patient generally have painful paresthesias., thalamic stroke aptients have dystonia-chorea and athetosis (not seen in this patient), thalamic lesion NEVER involves all sensory modalities., thalamic lesion involes only proprioception.
http://brain.oxfordjournals.org/cgi/cont.../124/2/299
your article says" An infarction or haemorrhage thalamic lesion can develop somatosensory disturbances and/or central pain in the opposite hemibody, analgesic or purely algesic thalamic syndrome characterised by contralateral anaesthesia (or hypaesthesia), contralateral weakness, ataxia and, often, persistent spontaneous pain."
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whew! this ws some heavy-duty exchanges......each stronger than the other.
iam still backing thalamus as the source, albeit weakly. any more inputs?
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DEJERAINE ROUSY SYNDROME - THALAMIC PAIN SYNDROME
METASTASIS ALWAYS AT WHITE GREY MATTER JN . HERE VERY PECULIAR METASTASIS
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ans.c bc of sensory invol.
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