05-26-2011, 01:46 PM
Welcome....,Please choose and answer the Qs in order. We are always looking for more volunteers. ... Any help would be appreciated .Thank you.
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This post is made again and please answer the Qs with more explanation,Thank you
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immune - 05/26/11 12:49
Q 1 ans is F since TB is a reportable disease
2.CC
When blood sugar is low, glucagon signals the adipocytes to activate hormone-sensitive lipase, and to convert triglycerides into free fatty acids
3.EE
Ninety percent of patients with gout develop excess urate stores due to an inability to excrete sufficient amounts of normally produced uric acid in the urine (underexcretion). The remaining patients either overconsume purines or produce excessive amounts of uric acid endogenously (overproduction).
•In rare cases, overproduction of uric acid is primary, due to a genetic disorder. These disorders include hypoxanthine-guanine phosphoribosyltransferase deficiency (Lesch-Nyhan syndrome), glucose-6-phosphatase deficiency (von Gierke disease), fructose 1-phosphate aldolase deficiency, and PP-ribose-P synthetase variants.
•Overproduction of uric acid may also occur in disorders that cause high cell turnover with release of purines, such as meat intake and seafood intake. These disorders include myeloproliferative and lymphoproliferative disorders, psoriasis, chemotherapy (tissue lysis), hemolytic anemias, pernicious anemia, ineffective erythropoiesis (as in B-12 deficiency), excessive exercise, and obesity.
•Overproduction of uric acid can occur from overconsumption of foods high in purines.
•Common causes of secondary gout due to underexcretion of uric acid include renal insufficiency, lead nephropathy (saturnine gout), starvation or dehydration, hypothyroidism, hyperparathyroidism, drugs (including loop and thiazide diuretics and cyclosporine A), low-dose aspirin, and chronic ethanol (especially beer and hard liquor) abuse. These disorders should be identified and corrected, if possible.
•Comorbidities, including hypertension, diabetes, renal insufficiency, hypertriglyceridemia, hypercholesterolemia, diabetes, obesity, and early menopause, are associated with a higher incidence of gout.
•Consumption of fructose-rich foods and beverages are associated with an increased risk of gout in both men and women
Because of no clinical symptoms or history we should think about risk factors that can cause Gout...
....gender...Men more than women
.......................age.....elderly more than younger
please clarify the answer,ty
4.CC
Gardenella vaginalis....gram varieble rod...cause vaginosis
white/gray vaginal discharge
Whiff test...add KOH...fishy smell
nonpainful
anaerobe
overgrowth of bacteria in vagina
Clue cells or vaginal epithelial cell covered with bacteria
treatment....Metronidazole
5.A
ans is A anterior white comissure as the clinical scenario sounds like syringomyelia which affect mostly the cervical spinal cord.
posted by immune
to add
crossing fiber of spinothalamic tract dameged...bilateral loss of pain and temprature sensation
6.CC
Lavastatin....-statins
HMG-CoA reductase inhibitors...dec.LDL..Bad cholestrol...mild eefect on HDL and triglycerides...inc.
mechanism of action....
1.inhibit cholestrol precursor,mevalonate
2.synthesizing LDL receptors
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This post is made again and please answer the Qs with more explanation,Thank you
********************************************************************************************
immune - 05/26/11 12:49
Q 1 ans is F since TB is a reportable disease
2.CC
When blood sugar is low, glucagon signals the adipocytes to activate hormone-sensitive lipase, and to convert triglycerides into free fatty acids
3.EE
Ninety percent of patients with gout develop excess urate stores due to an inability to excrete sufficient amounts of normally produced uric acid in the urine (underexcretion). The remaining patients either overconsume purines or produce excessive amounts of uric acid endogenously (overproduction).
•In rare cases, overproduction of uric acid is primary, due to a genetic disorder. These disorders include hypoxanthine-guanine phosphoribosyltransferase deficiency (Lesch-Nyhan syndrome), glucose-6-phosphatase deficiency (von Gierke disease), fructose 1-phosphate aldolase deficiency, and PP-ribose-P synthetase variants.
•Overproduction of uric acid may also occur in disorders that cause high cell turnover with release of purines, such as meat intake and seafood intake. These disorders include myeloproliferative and lymphoproliferative disorders, psoriasis, chemotherapy (tissue lysis), hemolytic anemias, pernicious anemia, ineffective erythropoiesis (as in B-12 deficiency), excessive exercise, and obesity.
•Overproduction of uric acid can occur from overconsumption of foods high in purines.
•Common causes of secondary gout due to underexcretion of uric acid include renal insufficiency, lead nephropathy (saturnine gout), starvation or dehydration, hypothyroidism, hyperparathyroidism, drugs (including loop and thiazide diuretics and cyclosporine A), low-dose aspirin, and chronic ethanol (especially beer and hard liquor) abuse. These disorders should be identified and corrected, if possible.
•Comorbidities, including hypertension, diabetes, renal insufficiency, hypertriglyceridemia, hypercholesterolemia, diabetes, obesity, and early menopause, are associated with a higher incidence of gout.
•Consumption of fructose-rich foods and beverages are associated with an increased risk of gout in both men and women
Because of no clinical symptoms or history we should think about risk factors that can cause Gout...
....gender...Men more than women
.......................age.....elderly more than younger
please clarify the answer,ty
4.CC
Gardenella vaginalis....gram varieble rod...cause vaginosis
white/gray vaginal discharge
Whiff test...add KOH...fishy smell
nonpainful
anaerobe
overgrowth of bacteria in vagina
Clue cells or vaginal epithelial cell covered with bacteria
treatment....Metronidazole
5.A
ans is A anterior white comissure as the clinical scenario sounds like syringomyelia which affect mostly the cervical spinal cord.
posted by immune
to add
crossing fiber of spinothalamic tract dameged...bilateral loss of pain and temprature sensation
6.CC
Lavastatin....-statins
HMG-CoA reductase inhibitors...dec.LDL..Bad cholestrol...mild eefect on HDL and triglycerides...inc.
mechanism of action....
1.inhibit cholestrol precursor,mevalonate
2.synthesizing LDL receptors